전체 논문 (180편 · 1/6)
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RFC5 enhances DNA damage response and immune escape via suppressing the cGAS-STING pathway in nasopharyngeal carcinoma.
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N-α-Acetyltransferase 40 promotes oral squamous cell carcinoma progression by enhancing FEN1 transcription and suppressing CD8 T cell antitumor immunity.
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CLDN18.2 in lung adenocarcinoma as a promising target of chimeric antigen receptor T cells.
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USP9X promotes acute myeloid leukemia progression and adriamycin resistance by regulating METTL7A deubiquitination.
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Roxadustat induces erythroid differentiation of erythroleukemia cells through the hypoxia inducible factor-α/GATA binding protein 1 axis.
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Fenofibrate anti-RARα/RXRα dimerization to attenuate all-trans retinoic acid-induced hyperlipidemia and hepatic steatosis in mice.
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UFL1 deficiency impairs skeletal muscle development by activating PERK/eIF2α/ATF4/CHOP pathway-dependent apoptosis.
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The function and molecular mechanism of HIF-1α interacted with p-STAT3 in promoting G6PD overexpression in chronic myelogenous leukemia cells.
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Protein 4.1R regulates CCDC26 and impacts myeloid leukemia progression.
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Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cell Signal, 135 (2025), 112027].
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OCIAD2-IQGAP1 interaction promotes pancreatic cancer progression by suppressing oxidative stress and mitochondria-mediated apoptosis.
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LRP1 regulates GDNF secretion and perineural invasion in pancreatic cancer via PI3K/AKT signaling.
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Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cellular Signalling 135 (2025) 112027].
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DPP4 suppresses pancreatic cancer growth by enhancing ferroptosis sensitivity through stabilization of ACSL4.
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The YAP1 and EPHA3 receptor tyrosine kinase axis regulates cellular plasticity and treatment response.
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Cellular signaling and radioresistance in prostate cancer.
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PGM5 - AS1 regulates ferroptosis - Mediated gastric cancer cell behavior via ERK signaling pathway.
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Transcriptional and post-transcriptional convergence of HES1 and IGF2BP2 sustains CSF2-dependent metabolic adaptability in gastric Cancer.
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METTL3-mediated m6A modification of circCDYL promotes gastric cancer progression by acting as miR-378a-5p sponge to regulate USP13 expression.
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M6A reader IGF2BP3 stabili1zes SLC7A11 to antagonize ferroptosis and promote gastric tumorigenesis.
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tRF-Ser-TGA-011 impedes gastric cancer progression by targeting the MAP3K13/JNK signaling cascade.
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The snoRNA SNORA21 promotes gastric tumorigenesis by attenuating P53 activity through CHK1 phosphorylation inhibition and PERP-dependent feedback loops.
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SRY-box transcription factor 7 functions as a tumor suppressor regulating Wnt signaling and cancer stemness with its promoter methylation as a potential biomarker.
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RARG promotes gastric cancer progression by activating PMEPA1 transcription and Wnt/β-catenin signalling.
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LINC01106 drives gastric cancer progression and tumor immune microenvironment remodeling via the miR-361-3p/DTL axis.
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GNE-493 suppresses gastric cancer development by targeting NAT10-mediated ac4C modification of HK2.
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Corrigendum to "Ginsenoside inhibits aerobic glycolysis in hepatocellular carcinoma through the hsa-miR-139-5p/AURKA axis" [Cellular Signalling 136 (2025) 112177].
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NCAPG confers ferroptosis resistance of hepatocellular carcinoma through NSUN2-mediated m5C modification.
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Hepatic stellate cells shape the ECM-disorganized and immunosuppressive microenvironment via CCL11/CCR3 axis in lenvatinib-treated hepatocellular carcinoma.
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CDS1 deficiency promotes colorectal cancer progression by suppressing CDP-DAG-induced ferroptosis in MDSCs.