Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cellular Signalling 135 (2025) 112027].
APA
Ding F, Guo Y, et al. (2026). Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cellular Signalling 135 (2025) 112027].. Cellular signalling, 112467. https://doi.org/10.1016/j.cellsig.2026.112467
MLA
Ding F, et al.. "Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cellular Signalling 135 (2025) 112027].." Cellular signalling, 2026, pp. 112467.
PMID
41807160
같은 제1저자의 인용 많은 논문 (5)
- Overlapping Pretarsal and Preseptal Orbicularis Oculi Muscles Enhance Pretarsal Fullness: Personalized Lower Eyelid Blepharoplasty for Asian Patients.
- Predictive model development and validation of functional liver imaging score for prognosis of patients with hepatocellular carcinoma after surgical resection: a multicenter study.
- Corrigendum to "High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway" [Cell Signal, 135 (2025), 112027].
- High glucose-induced mitochondrial fission drives pancreatic cancer progression through the H3K18la/TTK/BUB1B signal pathway.
- Abnormal levels of miRNA in pancreatic cancer are linked to tumor progression by regulating the translation of tumor-associated mRNA.