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Different Diseases, Different Escapes: Trastuzumab Deruxtecan Resistance in HER2-Amplified versus HER2-Low Breast Cancer.

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Cancer discovery 2026 Vol.16(2) p. 195-197
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O'Meara TA, Tarantino P

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In this issue of Cancer Discovery, Chen and colleagues demonstrate that, in preclinical models, HER2 expression level directly affects trastuzumab deruxtecan internalization and cytotoxicity, with cli

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APA O'Meara TA, Tarantino P (2026). Different Diseases, Different Escapes: Trastuzumab Deruxtecan Resistance in HER2-Amplified versus HER2-Low Breast Cancer.. Cancer discovery, 16(2), 195-197. https://doi.org/10.1158/2159-8290.CD-25-2102
MLA O'Meara TA, et al.. "Different Diseases, Different Escapes: Trastuzumab Deruxtecan Resistance in HER2-Amplified versus HER2-Low Breast Cancer.." Cancer discovery, vol. 16, no. 2, 2026, pp. 195-197.
PMID 41645714

Abstract

In this issue of Cancer Discovery, Chen and colleagues demonstrate that, in preclinical models, HER2 expression level directly affects trastuzumab deruxtecan internalization and cytotoxicity, with clinical data revealing divergent target dynamics depending on whether HER2 functions as an oncogenic driver or a dispensable antigen. Together with prior preclinical and clinical evidence, these findings support a context-dependent model in which target downregulation predominates in HER2-low disease, whereas payload resistance or rare binding-site mutations may dominate resistance in HER2-addicted tumors, with important implications for antibody-drug conjugate selection and sequencing. See related article by Chen et al., p. 235.

MeSH Terms

Humans; Breast Neoplasms; Erb-b2 Receptor Tyrosine Kinases; Drug Resistance, Neoplasm; Female; Trastuzumab; Immunoconjugates; Camptothecin; Antineoplastic Agents, Immunological; Antibodies, Monoclonal, Humanized; Gene Amplification