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Glucose metabolism in anterior pituitary adenomas.

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Clinica chimica acta; international journal of clinical chemistry 📖 저널 OA 0% 2023: 0/1 OA 2024: 0/1 OA 2025: 0/11 OA 2026: 0/106 OA 2023~2026 2026 Vol.578() p. 120587
Retraction 확인
출처

PICO 자동 추출 (휴리스틱, conf 2/4)

유사 논문
P · Population 대상 환자/모집단
환자: secretory pituitary adenomas
I · Intervention 중재 / 시술
추출되지 않음
C · Comparison 대조 / 비교
추출되지 않음
O · Outcome 결과 / 결론
Excessive TSH leads to increased T3 and T4 production, which in turn stimulate glucose uptake in muscle and enhance both glycolysis and gluconeogenesis. This review summarizes the current understanding of how hormone hypersecretion in pituitary adenomas contributes to glucose metabolism dysregulation.

Hernández-Brito V, Casco-Morales SL, Vega-Rosas A

ℹ️ 이 논문은 무료 전문이 아직 없습니다. 코퍼스 전체의 43.8%는 무료 가능 (통계 →) · 🏥 기관 EZproxy로 시도

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Glucose metabolism alterations are frequently observed in patients with secretory pituitary adenomas.

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↓ .bib ↓ .ris
APA Hernández-Brito V, Casco-Morales SL, Vega-Rosas A (2026). Glucose metabolism in anterior pituitary adenomas.. Clinica chimica acta; international journal of clinical chemistry, 578, 120587. https://doi.org/10.1016/j.cca.2025.120587
MLA Hernández-Brito V, et al.. "Glucose metabolism in anterior pituitary adenomas.." Clinica chimica acta; international journal of clinical chemistry, vol. 578, 2026, pp. 120587.
PMID 40912478 ↗

Abstract

Glucose metabolism alterations are frequently observed in patients with secretory pituitary adenomas. The most commonly secreted hormones in these tumors include prolactin, growth hormone (GH), adrenocorticotropic hormone (ACTH), and thyroid-stimulating hormone (TSH), all of which can disrupt glucose homeostasis through distinct pathophysiological mechanisms. Prolactin stimulates pancreatic β-cell proliferation, enhances insulin gene transcription, increases intracellular insulin content, and augments glucose-induced insulin secretion. GH promotes lipolysis and hepatic glucose production while impairing peripheral glucose uptake, contributing to insulin resistance. ACTH, via excess glucocorticoids, reduces GLUT-4 translocation, enhances gluconeogenesis and proteolysis, and inhibits glycogen synthesis. Excessive TSH leads to increased T3 and T4 production, which in turn stimulate glucose uptake in muscle and enhance both glycolysis and gluconeogenesis. This review summarizes the current understanding of how hormone hypersecretion in pituitary adenomas contributes to glucose metabolism dysregulation.

🏷️ 키워드 / MeSH 📖 같은 키워드 OA만

🏷️ 같은 키워드 · 무료전문 — 이 논문 MeSH/keyword 기반