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PAI-1, hematopoietic stem cell, and chronic myeloid leukemia.

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Biomedical journal 2026 Vol.49(1) p. 100854
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Takahashi N, Harigae H

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The new treatment goal for chronic myeloid leukemia (CML) is treatment-free remission, which requires the achievement and maintenance of a deep molecular response (DMR) using a tyrosine kinase inhibit

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APA Takahashi N, Harigae H (2026). PAI-1, hematopoietic stem cell, and chronic myeloid leukemia.. Biomedical journal, 49(1), 100854. https://doi.org/10.1016/j.bj.2025.100854
MLA Takahashi N, et al.. "PAI-1, hematopoietic stem cell, and chronic myeloid leukemia.." Biomedical journal, vol. 49, no. 1, 2026, pp. 100854.
PMID 40254273

Abstract

The new treatment goal for chronic myeloid leukemia (CML) is treatment-free remission, which requires the achievement and maintenance of a deep molecular response (DMR) using a tyrosine kinase inhibitor (TKI). However, only a limited number of patients achieve or maintain DMR. TM5614, a PAI1 inhibitor, can bring quiescent hematopoietic stem cells from the bone marrow niche into the cell cycle. Thus, the combination of TM5614 and TKI may eradicate CML cells. Currently, a phase III clinical trial is confirming the efficacy of TM5614 and TKI combination therapy.

MeSH Terms

Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Hematopoietic Stem Cells; Plasminogen Activator Inhibitor 1; Protein Kinase Inhibitors

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