본문으로 건너뛰기
← 뒤로

Clinical Impact and Clinicopathologic Correlations of Oncotype DX in Hormone Receptor-Positive/HER2-Negative Early Breast Cancer: Real-World Evidence From a Large Brazilian Cohort (GBECAM 0520).

JCO oncology practice 2026 p. OP2500899

Oliveira LJC, Araújo DC, de Araújo JAP, Rosa DD, Katz A, Assad-Suzuki D, Argolo D, Sanches SM, Testa L, Bines J, Kaliks RA, Gagliato DM, Barroso-Sousa R, Corrêa TS, Shimada AK, Dos Anjos CH, Linck R, Megid TBC, Batista DN, Gomes DM, Cesca MG, Gaudêncio D, Moura LMA, Bonadio RC, de Souza ZS, Beal JR, Lopes MM, Sales LT, Parisi Marlière JLF, Mano MS

📝 환자 설명용 한 줄

[PURPOSE] Oncotype DX (ODX) is a validated gene expression assay that provides prognostic and predictive information to guide adjuvant chemotherapy (ACT) decisions in hormone receptor-positive, HER2-n

🔬 핵심 임상 통계 (초록에서 자동 추출 — 원문 검증 권장)
  • 추적기간 4.8 years

이 논문을 인용하기

BibTeX ↓ RIS ↓
APA Oliveira LJC, Araújo DC, et al. (2026). Clinical Impact and Clinicopathologic Correlations of Oncotype DX in Hormone Receptor-Positive/HER2-Negative Early Breast Cancer: Real-World Evidence From a Large Brazilian Cohort (GBECAM 0520).. JCO oncology practice, OP2500899. https://doi.org/10.1200/OP-25-00899
MLA Oliveira LJC, et al.. "Clinical Impact and Clinicopathologic Correlations of Oncotype DX in Hormone Receptor-Positive/HER2-Negative Early Breast Cancer: Real-World Evidence From a Large Brazilian Cohort (GBECAM 0520).." JCO oncology practice, 2026, pp. OP2500899.
PMID 41791005
DOI 10.1200/OP-25-00899

Abstract

[PURPOSE] Oncotype DX (ODX) is a validated gene expression assay that provides prognostic and predictive information to guide adjuvant chemotherapy (ACT) decisions in hormone receptor-positive, HER2-negative (HR+/HER2-) early breast cancer (eBC), including node-negative (N0) and 1-3 node-positive (N1) disease. In Brazil, limited access to gene expression signatures (GES) remains a barrier to individualized treatment decisions.

[METHODS] We conducted a multicenter retrospective study across nine Brazilian private cancer centers to evaluate clinicopathologic predictors of high genomic risk (ODX recurrence score [RS] >25) and to assess the clinical impact of ODX on ACT decision making.

[RESULTS] Between 2005 and 2024, 935 patients were included, with a notably higher representation of young (≤40 years: 10.9%) and premenopausal (40.7%) women than typically reported in randomized clinical trials. Ki-67 >20%, progesterone receptor (PR) expression ≤30%, and histologic grade 3 were independently associated with high RS in both univariable and multivariable analyses (all <.001), with Ki-67 emerging as the strongest clinicopathologic predictor. Overall, ODX testing was associated with an estimated 12.6% absolute reduction in ACT recommendations, with substantial impact among postmenopausal patients with N1 disease (94% absolute reduction) and N0 patients older than 50 years with high clinical risk (65.2% absolute reduction). With a median follow-up of 4.8 years, the estimated 5-year real-world invasive disease-free survival and distant disease-free survival were 100% and 100% for those with RS <11, 98.2% and 99.4% for RS 11-25, and 90.9% and 92.5% for RS >25, respectively.

[CONCLUSION] In this data set, ODX demonstrated utility in guiding ACT decision making and supporting personalized treatment by reducing both overtreatment and undertreatment. In resource-constrained settings, surrogate markers such as Ki-67, PR expression, and histologic grade may serve as practical tools to guide risk-adapted clinical decisions.