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Androgens and alopecia.

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Molecular and cellular endocrinology 📖 저널 OA 22.9% 2021: 2/3 OA 2022: 0/4 OA 2023: 0/6 OA 2024: 0/11 OA 2025: 5/15 OA 2026: 8/23 OA 2021~2026 2002 Vol.198(1-2) p. 89-95 피인용 52회 cited 225 RCR 4.44 Hair Growth and Disorders
TL;DR It is suggested that MPHL and FPHL are distinct clinical entities, with disparate pathophysiologies, and studies that elucidate the molecular mechanisms by which androgens regulate hair growth would provide greater understanding of these differences.
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PubMed DOI OpenAlex Semantic 마지막 보강 2026-05-12
📑 코퍼스 인용 관계 · 인용됨 52
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연도별 인용 (2012–2026) · 합계 186
OpenAlex 토픽 · Hair Growth and Disorders Historical Gender and Feminism Studies

Kaufman KD

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Abstract

Androgens have profound effects on scalp and body hair in humans. Scalp hair grows constitutively in the absence of androgens, while body hair growth is dependent on the action of androgens. Androgenetic alopecia, referred to as male pattern hair loss (MPHL) in men and female pattern hair loss (FPHL) in women, is due to the progressive miniaturization of scalp hair. Observations in both eunuchs, who have low levels of testicular androgens, and males with genetic 5alpha-reductase (5alphaR) deficiency, who have low levels of dihydrotestosterone (DHT), implicate DHT as a key androgen in the pathogenesis of MPHL in men. The development of finasteride, a type 2-selective 5alphaR inhibitor, further advanced our understanding of the role of DHT in the pathophysiology of scalp alopecia. Controlled clinical trials with finasteride demonstrated improvements in scalp hair growth in treated men associated with reductions in scalp DHT content, and a trend towards reversal of scalp hair miniaturization was evident by histopathologic evaluation of scalp biopsies. In contrast to its beneficial effects in men, finasteride did not improve hair growth in postmenopausal women with FPHL. Histopathological evaluation of scalp biopsies confirmed that finasteride treatment produced no benefit on scalp hair in these women. These findings suggest that MPHL and FPHL are distinct clinical entities, with disparate pathophysiologies. Studies that elucidate the molecular mechanisms by which androgens regulate hair growth would provide greater understanding of these differences.
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It is suggested that MPHL and FPHL are distinct clinical entities, with disparate pathophysiologies, and studies that elucidate the molecular mechanisms by which androgens regulate hair growth would p

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↓ .bib ↓ .ris
APA 7 Kaufman, K. D. (2002). Androgens and alopecia.. Molecular and cellular endocrinology, 198(1-2), 89-95. https://doi.org/10.1016/s0303-7207(02)00372-6
Vancouver Kaufman KD. Androgens and alopecia. Mole. cell. endo.. 2002;198(1-2):89-95. doi:10.1016/s0303-7207(02)00372-6
AMA 11 Kaufman KD. Androgens and alopecia. Mole. cell. endo.. 2002;198(1-2):89-95. doi:10.1016/s0303-7207(02)00372-6
Chicago Kaufman, K. D.. 2002. "Androgens and alopecia." Molecular and cellular endocrinology 198 (1-2): 89-95. https://doi.org/10.1016/s0303-7207(02)00372-6
MLA 9 Kaufman, K. D.. "Androgens and alopecia." Molecular and cellular endocrinology, vol. 198, no. 1-2, 2002, pp. 89-95. doi:10.1016/s0303-7207(02)00372-6.
PMID 12573818 ↗
DOI 10.1016/s0303-7207(02)00372-6

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그래프 OA 노드: 8/8 (100%) · 참조 0편 · 후속 8편

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