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From actinic keratosis to squamous cell carcinoma: pathophysiology revisited.

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Journal of the European Academy of Dermatology and Venereology : JEADV 📖 저널 OA 28.3% 2021: 14/53 OA 2022: 9/54 OA 2023: 21/68 OA 2024: 21/48 OA 2025: 48/75 OA 2026: 32/66 OA 2021~2026 2017 Vol.31 Suppl 2() p. 5-7 피인용 1회 cited 171 OA RCR 6.20 Nonmelanoma Skin Cancer Studies
TL;DR It is proposed that all AKs, regardless of the grade, should be carefully monitored and appropriately treated in clinical practice and the ideal treatment should be able to eradicate AK lesions and reverse the underlying field cancerization.
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PubMed DOI OpenAlex Semantic 마지막 보강 2026-05-08
연도별 인용 (2015–2026) · 합계 171
OpenAlex 토픽 · Nonmelanoma Skin Cancer Studies Cutaneous lymphoproliferative disorders research Cutaneous Melanoma Detection and Management

Fernandez Figueras MT

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Abstract

The precursor of most cutaneous invasive squamous cell carcinomas (iSCCs) is intraepithelial UV-induced damage, known as field cancerization, which can eventually transform into actinic keratosis (AK). Although AK is the most common precursor of iSCC, many AKs will either persist in the same stage or regress, while only a few will progress into iSCC. Nevertheless, because the progression of individual AKs cannot be predicted, it has been proposed that all AKs, regardless of the grade, should be carefully monitored and appropriately treated in clinical practice. Modern imaging techniques such as dermatoscopy, reflectance confocal microscopy (RCM) and high-definition optical coherence tomography (HD-OCT) may have potential to monitor the evolution of actinic field damage. Dermatoscopy can be used to differentiate between AK, intraepidermal carcinoma (IEC) and SCC which may help clinicians to diagnose in situ or invasive lesions at an earlier stage. HD-OCT and RCM can be used to detect cellular and histological changes characteristic of subclinical lesions, allowing visualization of previously invisible lesions. As development of invasive AK directly from the cancer field cannot be ruled out, the ideal treatment should be able to eradicate AK lesions and reverse the underlying field cancerization.
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It is proposed that all AKs, regardless of the grade, should be carefully monitored and appropriately treated in clinical practice and the ideal treatment should be able to eradicate AK lesions and re

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↓ .bib ↓ .ris
APA 7 MT, F. F. (2017). From actinic keratosis to squamous cell carcinoma: Pathophysiology revisited.. Journal of the European Academy of Dermatology and Venereology : JEADV, 31 Suppl 2, 5-7. https://doi.org/10.1111/jdv.14151
Vancouver MT FF. From actinic keratosis to squamous cell carcinoma: pathophysiology revisited. Jour. Euro. Acad. Derm. Vene. : JEAD.. 2017;31 Suppl 2:5-7. doi:10.1111/jdv.14151
AMA 11 MT FF. From actinic keratosis to squamous cell carcinoma: pathophysiology revisited. Jour. Euro. Acad. Derm. Vene. : JEAD.. 2017;31 Suppl 2:5-7. doi:10.1111/jdv.14151
Chicago MT, F. F.. 2017. "From actinic keratosis to squamous cell carcinoma: pathophysiology revisited." Journal of the European Academy of Dermatology and Venereology : JEADV 31 Suppl 2: 5-7. https://doi.org/10.1111/jdv.14151
MLA 9 MT, F. F.. "From actinic keratosis to squamous cell carcinoma: pathophysiology revisited." Journal of the European Academy of Dermatology and Venereology : JEADV, vol. 31 Suppl 2, 2017, pp. 5-7. doi:10.1111/jdv.14151.
PMID 28263020 ↗
DOI 10.1111/jdv.14151

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