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Degrade to Display: Coupling Checkpoint Degradation with Exogenous Antigen Presentation to Boost Antitumor Immunity.

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ACS chemical biology 2026 CAR-T cell therapy research
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PubMed DOI OpenAlex 마지막 보강 2026-04-29
OpenAlex 토픽 · CAR-T cell therapy research Nanoplatforms for cancer theranostics Cancer Immunotherapy and Biomarkers

Ma Y, Jiang J, Han Y

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Membrane and extracellular targeted protein degradation (meTPD) has emerged as a revolutionary therapeutic modality against cancer, enabling the lysosomal clearance of traditionally undruggable oncoge

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↓ .bib ↓ .ris
APA Yicong Ma, Jianfei Jiang, Yu Han (2026). Degrade to Display: Coupling Checkpoint Degradation with Exogenous Antigen Presentation to Boost Antitumor Immunity.. ACS chemical biology. https://doi.org/10.1021/acschembio.6c00191
MLA Yicong Ma, et al.. "Degrade to Display: Coupling Checkpoint Degradation with Exogenous Antigen Presentation to Boost Antitumor Immunity.." ACS chemical biology, 2026.
PMID 42023934 ↗

Abstract

Membrane and extracellular targeted protein degradation (meTPD) has emerged as a revolutionary therapeutic modality against cancer, enabling the lysosomal clearance of traditionally undruggable oncogenic targets. However, the therapeutic potential of the current meTPD method is frequently compromised by its reliance on loss-of-function elimination in the face of tumor immunosuppression and low immunogenicity. This In Focus article highlights the development of the intratumoral vaccination chimera (iVAC), a "meTPD-Plus" platform that advances beyond target elimination to actively reprogram tumor immunogenicity. By coupling the degradation of PD-L1 with the cross-presentation of exogenous antigens within tumors, iVAC chemically reprograms cancer cells into pseudo antigen-presenting cell entities (pseudo-APCs) capable of engaging bystander memory T cells. This dual-mechanism approach simultaneously relieves checkpoint-mediated immune suppression and enhances tumor antigen presentation, thereby redirecting viral-specific bystander T cells toward tumor recognition and establishing durable, systemic antitumor immunity.

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