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Per- and polyfluoroalkyl substance concentrations during pregnancy and at birth and risk of childhood acute lymphoblastic leukemia.

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Environmental research 📖 저널 OA 28.6% 2022: 1/3 OA 2023: 0/1 OA 2025: 1/8 OA 2026: 8/22 OA 2022~2026 2025 Vol.285(Pt 3) p. 122436
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Morimoto LM, Metayer C, Dolios G, Wiemels JL, Ma X, Guan H

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[BACKGROUND] Per- and polyfluoroalkyl substances (PFAS) comprise a class of persistent environmental pollutants with potential carcinogenic effects, but their impact on childhood cancer remains undere

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APA Morimoto LM, Metayer C, et al. (2025). Per- and polyfluoroalkyl substance concentrations during pregnancy and at birth and risk of childhood acute lymphoblastic leukemia.. Environmental research, 285(Pt 3), 122436. https://doi.org/10.1016/j.envres.2025.122436
MLA Morimoto LM, et al.. "Per- and polyfluoroalkyl substance concentrations during pregnancy and at birth and risk of childhood acute lymphoblastic leukemia.." Environmental research, vol. 285, no. Pt 3, 2025, pp. 122436.
PMID 40716601 ↗

Abstract

[BACKGROUND] Per- and polyfluoroalkyl substances (PFAS) comprise a class of persistent environmental pollutants with potential carcinogenic effects, but their impact on childhood cancer remains underexplored. A child's exposure to PFAS can occur through various pathways postnatally, including contaminated food, water, and consumer products; and in utero, as PFAS can cross the placenta.

[METHODS] To investigate the association between early-life PFAS exposure and the risk of childhood acute lymphoblastic leukemia (ALL), we analyzed archived blood samples from children diagnosed with ALL and matched cancer-free controls. Using novel untargeted liquid chromatography-high resolution mass spectrometry (LC-HRMS), we measured PFAS levels in paired maternal pregnancy and child newborn blood samples.

[RESULTS] Our study identified an independent association between MeFOSAA levels at birth and increased ALL risk, particularly among children diagnosed at 2 years of age or younger. MeFOSAA measured in maternal second-trimester blood showed a weak association with ALL, although it was not statistically significant.

[CONCLUSIONS] These results suggest that early-life exposure to MeFOSAA may play a critical role in the development of childhood ALL. Our findings corroborate previous reports linking MeFOSAA exposure during pregnancy to childhood ALL, highlighting its potential carcinogenicity during key developmental windows.

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