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Identifying genes and pathways in familial lymphoid cancers using whole exome sequencing.

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Leukemia & lymphoma 📖 저널 OA 9.2% 2022: 1/1 OA 2025: 2/55 OA 2026: 15/137 OA 2022~2026 2026 Vol.67(5) p. 1014-1028 OA Wnt/β-catenin signaling in developme
TL;DR It is hypothesized that there are shared susceptibility factors in families with these heterogenous lymphoid malignancies and identifying factors predisposing to different types of lymphoid cancers will help understand the etiology of these neoplasms.
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PubMed DOI OpenAlex Semantic 마지막 보강 2026-04-30
OpenAlex 토픽 · Wnt/β-catenin signaling in development and cancer Lymphoma Diagnosis and Treatment Kruppel-like factors research

Ralli S, Jones SJ, Leach S, Lynch HJ, Brooks-Wilson AR

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It is hypothesized that there are shared susceptibility factors in families with these heterogenous lymphoid malignancies and identifying factors predisposing to different types of lymphoid cancers wi

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APA Sneha Ralli, Samantha Jones, et al. (2026). Identifying genes and pathways in familial lymphoid cancers using whole exome sequencing.. Leukemia & lymphoma, 67(5), 1014-1028. https://doi.org/10.1080/10428194.2026.2624656
MLA Sneha Ralli, et al.. "Identifying genes and pathways in familial lymphoid cancers using whole exome sequencing.." Leukemia & lymphoma, vol. 67, no. 5, 2026, pp. 1014-1028.
PMID 41652927 ↗

Abstract

Lymphoid cancers of different types and subtypes are known to cluster in families. We hypothesize that there are shared susceptibility factors in families with these heterogenous lymphoid malignancies. Exome sequencing was performed on 100 individuals from 43 lymphoid cancer pedigrees. Variants from 37 families were ranked using the eights-based vriant anking in edigrees (WARP) pipeline. Six affected unrelated probands were used for interpretation only. We detected recurrent variants in the germline lymphoid cancer gene in 4 (9%) of the 43 families, and variants in other genes involved in lymphoid cancers: and . Variants in genes including and involved in the WNT/β-catenin pathway were identified, representing a novel observation. Some variants appeared to segregate with specific types of lymphoid cancers; others were shared across different subtypes. Identifying factors predisposing to different types of lymphoid cancers will help understand the etiology of these neoplasms.

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