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Case Report: Durable complete response to olaparib in a patient with BRCA2-driven metastatic gastric adenocarcinoma after failure of chemo-immunotherapy.

증례보고 1/5 보강
Frontiers in oncology 📖 저널 OA 100% 2021: 15/15 OA 2022: 98/98 OA 2023: 60/60 OA 2024: 189/189 OA 2025: 1004/1004 OA 2026: 620/620 OA 2021~2026 2026 Vol.16() p. 1769043
Retraction 확인
출처

PICO 자동 추출 (휴리스틱, conf 3/4)

유사 논문
P · Population 대상 환자/모집단
환자: advanced gastric cancer
I · Intervention 중재 / 시술
gastrectomy with intraoperatively detected liver metastases
C · Comparison 대조 / 비교
추출되지 않음
O · Outcome 결과 / 결론
The patient achieved a complete metabolic response on FDG PET-CT, which has been sustained for more than three years under continuous olaparib therapy with acceptable tolerability. This case highlights the importance of molecularly guided treatment strategies and homologous recombination repair deficiency in selected patients with advanced gastric cancer.

Karakaya G

📝 환자 설명용 한 줄

Metastatic gastric adenocarcinoma is associated with poor prognosis and limited treatment options after failure of chemotherapy and immunotherapy.

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↓ .bib ↓ .ris
APA Karakaya G (2026). Case Report: Durable complete response to olaparib in a patient with BRCA2-driven metastatic gastric adenocarcinoma after failure of chemo-immunotherapy.. Frontiers in oncology, 16, 1769043. https://doi.org/10.3389/fonc.2026.1769043
MLA Karakaya G. "Case Report: Durable complete response to olaparib in a patient with BRCA2-driven metastatic gastric adenocarcinoma after failure of chemo-immunotherapy.." Frontiers in oncology, vol. 16, 2026, pp. 1769043.
PMID 42022316 ↗

Abstract

Metastatic gastric adenocarcinoma is associated with poor prognosis and limited treatment options after failure of chemotherapy and immunotherapy. Although BRCA1/2 alterations are well-established predictive biomarkers for PARP inhibitor efficacy in several malignancies, their role in gastric cancer remains incompletely defined. We report a 65-year-old male with metastatic gastric adenocarcinoma who underwent gastrectomy with intraoperatively detected liver metastases. The disease progressed following platinum-based chemotherapy combined with nivolumab and subsequent paclitaxel-ramucirumab therapy. Comprehensive genomic profiling using the Tempus xT assay identified a pathogenic germline BRCA2 frameshift mutation with somatic loss of heterozygosity. Based on this molecular profile, off-label olaparib therapy was initiated. The patient achieved a complete metabolic response on FDG PET-CT, which has been sustained for more than three years under continuous olaparib therapy with acceptable tolerability. This case highlights the importance of molecularly guided treatment strategies and homologous recombination repair deficiency in selected patients with advanced gastric cancer.

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