In situ laser fenestration of Metallic covered stent for the treatment of malignant Central Airway Stenosis: A case report.
Malignant central airway stenosis is a life-threatening complication of respiratory malignancies.
APA
He Y, Hu Z, et al. (2026). In situ laser fenestration of Metallic covered stent for the treatment of malignant Central Airway Stenosis: A case report.. Respiratory medicine case reports, 59, 102371. https://doi.org/10.1016/j.rmcr.2026.102371
MLA
He Y, et al.. "In situ laser fenestration of Metallic covered stent for the treatment of malignant Central Airway Stenosis: A case report.." Respiratory medicine case reports, vol. 59, 2026, pp. 102371.
PMID
41657859
Abstract
Malignant central airway stenosis is a life-threatening complication of respiratory malignancies. Self-expanding covered metal stents are an effective treatment, offering self-adaptability without the need for customization, making them suitable for emergency use. Y-shaped stents are preferred in most cases due to their low migration risk. However, they may cover the right upper lobe bronchus, leading to atelectasis, which requires intervention. This is the first reported case of central airway stenosis caused by bronchial metastasis from colon cancer. After placement of a Y-shaped covered stent, endobronchial modification using a YAG laser was performed to restore right upper lobe ventilation. The procedure successfully relieved the patient's dyspnea. This case provides clinical reference value, though further reports are needed to confirm its generalizability.
같은 제1저자의 인용 많은 논문 (5)
- ESE and Transfer Learning for Breast Tumor Classification.
- Home-based multimodal prehabilitation before colorectal cancer surgery: a systematic review and meta-analysis.
- Advances and challenges of ZIF-based nanocomposites in immunotherapy and anti-inflammatory therapy.
- Intravascular Large B-Cell Lymphoma Presenting With Urinary and Fecal Incontinence: A Case Report.
- Tanshinone IIA targets RNF123 to inhibit non-small cell lung cancer cell proliferation, migration and invasion via KAT2B-mediated H3K18ac modification.