📖 OA 비율이 비슷한 저널 — 이 저널 88.5% 기준 ±5pp
코퍼스 내 인용 상위 5
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Osimertinib-induced DNA resistance mutations in cerebrospinal fluid of epidermal growth factor receptor-mutated non-small-cell lung carcinoma patients developing leptomeningeal metastases: Osimertinib Resistance Analysis-leptomeningeal metastases study.
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Encorafenib and binimetinib followed by radiotherapy for patients with BRAFV600-mutant melanoma and brain metastases (E-BRAIN/GEM1802 phase II study).
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Targeting IGF2 to reprogram the tumor microenvironment for enhanced viro-immunotherapy.
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Cholesterol-metabolic tumor-associated macrophages regulate tumor budding-like cell subpopulation to promote chordoma stemness via BACH1/ANGPTL4/SDC4 axis.
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Phase II study of safusidenib erbumine in patients with chemotherapy- and radiotherapy-naïve isocitrate dehydrogenase 1-mutated WHO grade 2 gliomas.
전체 논문 (130편 · 1/5)
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Multi-Transcriptomics Analysis Identifies NNAT as a Key Molecule Driving the Invasive Phenotype Across All Lineages of Pituitary Adenomas.
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Single-nucleus transcriptomics, pharmacokinetics, and pharmacodynamics of CDK4/6 and mTOR inhibition in a Phase 0/1 trial of recurrent high-grade glioma.
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Combined Patch-clamp Electrophysiology and Single-Cell Genomic Analysis Reveal Spiking Tumor Cells at the Neocortical Glioblastoma Interface in Humans.
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Cysteine addiction in drug resistant glioblastoma and therapeutic targeting with designer selenium compounds.
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Nuclear export as a therapeutic vulnerability in ZFTA-RELA ependymoma.
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Molecular modulators of CDK4/6 inhibitor response in experimental glioma identified through genome-wide CRISPR-Cas9 screening.
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DNA damage response profile distinguishes poor-acting gliomas with shared methylome signatures.
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AI-driven WHO 2021 classification of gliomas based only on H&E-stained slides.
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lncRNA LUCAT1 Regulates DNA Damage Response in Glioma Stem Cells Under Hypoxia.
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DNA copy number patterns reveal prognostic markers and elucidate mechanisms of evolution in IDH-mutant astrocytoma.
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Single-cell profiling of peripheral and local immune compartments reveal unique genotype-independent prognostic immune signatures across isocitrate dehydrogenase-stratified glioma.
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YBX1&YBX3 as novel targets to potentiate immune checkpoint blockade response in gliomas.
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Interrogation of the cellular hierarchies reveals neoplastic evolution and therapeutic vulnerability in craniopharyngioma.
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Distinct tumor immune microenvironmental (TIME) landscapes drive divergent immunotherapy responses in glioblastoma.
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Integrated immune profiling of chordomas reveals spatially organized niches and functional heterogeneity.
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Super-Enhancer-Driven TCF4 Orchestrates Neuroblastoma Metastasis by Sphingolipid-Dependent Membrane Remodeling and ITGB1-FAK Activation.
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MIF-CD74 signaling drives immune modulation in medulloblastoma.
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Spatiotemporal Role of GLI2 in Driving SHH-Medulloblastoma Tumorigenesis.
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Oncolytic Zika virus therapy leverages CCR2+ monocytes to boost anti-glioblastoma T cell responses.
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Cholesterol-metabolic tumor-associated macrophages regulate tumor budding-like cell subpopulation to promote chordoma stemness via BACH1/ANGPTL4/SDC4 axis.
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DNA methylation profiling reveals a novel subtype harboring IDH mutation in H3-altered gliomas.
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Exploring the immune environment of glioblastoma in humanized mouse models.
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Meningioma cell reprogramming and microenvironment interactions underlie brain invasion.
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Laser Interstitial Thermal Therapy Enhances Bidirectional Blood-Brain Barrier Permeability in Glioblastoma.
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An integrated analysis of three medulloblastoma clinical trials refines risk-stratification approaches for reducing toxicity and improving survival.
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Single-nucleus transcriptomics of spinal ependymoma subtypes recognizes intratumoral heterogeneity.
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Multiple patient-derived glioblastoma models reveal synthetic lethality through concurrent PI3K and CDK4/6 inhibition by blocking trans-active cooperation.
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Phase I/II and window-of-opportunity study of pamiparib and metronomic temozolomide for recurrent isocitrate dehydrogenase mutant gliomas.
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Integrated transcriptomic landscape of medulloblastoma and ependymoma reveals novel tumor subtype-specific biology.
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Assessment of molecular tools in pediatric, adolescent, and young adult meningioma highlights the need for lifespan precision in neuro-oncology.