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Active RAC1 Promotes Tumorigenic Phenotypes and Therapy Resistance in Solid Tumors.
Acting as molecular switches, all three members of the Guanosine triphosphate (GTP)-ase-family, Ras-related C3 botulinum toxin substrate (RAC), Rho, and Cdc42 contribute to various processes of oncogenic transformations in several solid tum…